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Mental Health Blog

Thursday
May132010

How to Treat Insomnia or Getting Rid of Agrypniaphobia

General considerations

Excessive anxiety, being upset and tense, and having difficulty relaxing, as well as apprehensiveness about the future all interfere with restful sleep.

Rx: What are some of the treatments for insomnia?  Relaxation and stress-reduction are very effective but part of the solution depends on whether you are having trouble initiating sleep or staying asleep.

Of the former, limit caffeine and alcohol consumption in the evenings.  Eat regular and healthy meals and take a multivitamin supplement once-a-day.  Set your alarm and put the clock underneath your bed to avoid nervousness caused by clock-watching.

Use soothing music to fall asleep to, if that is helpful (e.g., quiet piano solos).  A hot bath before sleep is also very effective (particularly, when followed by a loving massage).  So are regular and satisfying sexual unions.

For the latter, use a contoured pillow to avoid stress on neck and turn over your mattress to get even wear (or consider getting a more comfortable mattress).  Set room temperature to 75 degrees F or below and keep humidity down; higher temperatures tend to disturb sleep, particularly, REM sleep and stages 3 and 4 (see below).

Keep noise down during sleep: Women are more sensitive to noise and noise sensitivity increases with age.  Only use sleeping pills for emergencies as they depend to depress REM and thus interfere with the quality of sleep.  They also cause rebound insomnia.  In general, psychoactive drugs are not effective in the treatment of insomnia, at least, not on a long-term basis.

A specific plan

Get up at the same time every morning and get regular exercise 3x-a-week in the morning, preferably.  One approach, if you have trouble initiating sleep, is to begin by going to bed four hours before you plan to get up.  And increase that by one half-hour a night for every five days of sleep in which you slept for at least 90% of the evening, an index of your “sleep efficiency” or the percentage of time you actually slept.  Continue this procedure until you are getting a full night’s sleep.  Generally, this is 8 – 8 1/2 hours but some people need less and some more.

Biology of sleep and dreaming

There are five stages of sleep that have been revealed through the use of electroencephalograms (EEG), eye movement recordings, and recordings of muscle activity.  Neurons (nerve cells) in the brain generate electrochemical signals that routinely spread across the cortex or outer layers the brain and produce an electrical field that can be measured and that manifest regular rhythms or “brain waves.”  They are measured by EEG.  Here are the five stages of sleep:

Awake 1: Eyes open – Beta waves (14-30 Hz)
Awake 2: Relaxed with eyes closed; Alpha waves (8-13 Hz)
Stage 1: Theta waves (4-7 Hz)
Stage 2: Bursts of high frequency waves (i.e., sleep spindles)
Stage 3: Delta waves (1-3 Hz)
Stage 4: Delta waves
REM: Rapid eye movement with theta waves

These stages cycle through 90” periods.  The first four stages are called non-REM sleep (NREM).  Disorders such as sleeptalking or sleepwalking occur in stages three and four (i.e., not when you are dreaming).  Night terrors, which may occur in children 3 – 8 years of age and during the first two hours of sleep, turn up in stage four.  They are believed to be due to faulty maturation of brain stem and also due to psychosocial stress.  Nighmares occur in REM sleep when dreaming occurs.  Surprisingly, when sleeping we do monitor the outside environment.  For example, mothers are particularly responsive to their infant’s nighttime cries.  Insomniacs appear to be completely unable to shutout outside stimuli and may be partly the cause of their sleep problems.  In any event, sleep problems are very common.  In the US, about 50% of adults report problems in initiating or staying asleep.  You are not alone.

Some suggesting reading:

Hauri, P., & Linde, S. (1990). No more sleepless nights. NY: Wiley.

Thursday
May132010

Stress Reduction Techniques

Meditation is a very effective technique for relieving psychosocial stress and has significant scientific support. Herbert Benson, M.D., at Harvard Medical School, has been promoting meditation for stress reduction since the early 1970s in his well-known book, The Relaxation Response. Here are some essential components of Dr. Benson’s relaxation response:

A quiet environment.

A comfortable position: You should start by closing your eyes and relaxing your muscles progressing from the feet to the head.  You should breathe slowly and naturally and use one of the mental devices below as you exhale.

A mental device: A sound, word, phrase or prayer repeatedly silently or aloud or with a fixed gaze on an object.

A passive attitude: Please do not worry about how well you are performing the technique and put aside distracting thoughts.

Practice the technique once or twice daily before breakfast and before dinner for 10-15 minutes.

You may also elicit the relaxation response while exercising.

Remember, there are two basic kinds of meditation. Either of these two are equally effective.

The path of concentration  (e.g., yoga, transcendental meditation, Sufism).  The mind focuses on specific external object.  For example, a mantra, a prayer, a picture, a candle flame, a spot in the lower abdomen, a bodily sensation, or a mandala.

The path of mindfulness (e.g., Krishamurti, Gurdjieff).  The mind observes itself.  For example, internal sensations, mental states, workings of the mind, breathing, position of the limbs, bodily states, or mood.

Hatha yoga as well as other forms of yoga are also effective for stress-reduction as is physical exercise and therapeutic massage. Hatha yoga involves, among other things, physical postures or asanas that are intended to promote physical well-being; improved flexibility, strength, and stamina; as well as encourage mental relaxation.  Hook-up with a yoga instructor in your neighborhood or purchase a yoga DVD.

Physical exercise: Vigorous physical activity has many important benefits.  Proper training (conditioning and technique), equipment, clothing, and footwear can reduce sports injuries.  Consider joining a health club or purchasing weights and equipment for home use.  A fitness instructor may be a good idea, initially, too.

Increases the number and size of blood vessels in heart and muscles
Increases elasticity of blood vessels
Increases efficiency of exercising muscles
Increases efficiency of the heart
Increases tolerance to stress
Decreases cholesterol and triglycerides
Lowers blood pressure reducing the risk of heart attack and stroke

Therapeutic massage: Therapeutic massage involves the manipulation of soft tissues of the body including skin, muscles, tendons, ligaments, and joints.  It reduces pain and psychosocial stress and may induce the relaxation response as well as reduce anxiety and depression.  It may also aid sleep and may have cardiovascular benefits.  Find a practitioner in your area.

Some suggested reading:

Benson, H. (1975).  The relaxation response. NY: HarperCollins.

Tuesday
Apr272010

What is Neuropsychology and Neurocognitive Rehabilitation?

Cognitive neuroscience is the study of how the central nervous system (brain and spinal cord) gives rise to higher-order cognitive functions such as attention, memory, language, and various intellective abilities such as planning and organizational abilities as well as mathematical and visual-spatial skills.  The clinical application of cognitive neuroscience is the field of clinical neuropsychology.

Clinical neuropsychology evaluates and assesses higher cognitive abilities that may have been compromised by disease or trauma to the brain such as multiple sclerosis, stroke, epilepsy, dementia, traumatic brain injury due to a traumatic external event to the brain (e.g., slipping on pavement and hitting one’s head) and other internal and external events that affect brain function.

The rehabilitation and remediation of brain function may occur through surgery, the use of various drugs (such as cholinesterase inhibitors in Alzheimer’s disease), as well as through the use of neurocognitive rehabilitation.  Neurocognitive rehabilitation or training involves the use cognitive training programs, often computer-based, to rehabilitate cognitive functions like attention, memory, and similar intellective abilities in individuals who have compromised higher-order cognitive abilities.

Clinical neuropsychology is also used to assess the effects of psychoactive drugs on higher-order cognitive functions as well as assess individuals ability to work, live independently, and the like. Neuropsychological assessment uses various mental tasks to assess higher-order cognitive functions in children (such as learning disabilities, problems in attention and concentration, and emotional issues), adolescents (such as depression and psychosis), and adults and the elderly (such as cerebrovascular disorders affecting higher-order cognitive functions, dementia, and compromised language abilities after stroke).

Tuesday
Apr272010

Psychosomatic Illness

Historical periods affect what physical and mental symptoms are expressed in illnesses having psychological causes.  Some medical professionals believe that the unconscious mind is the causative agent.

Physical symptoms having a psychogenic origin are common and the processes that cause them in the mind/body are known as “somatization.”  When there is actual physical disease but the patient’s responses are inappropriate or exaggerated, the processes that cause them in the mind/body are said to be somatogenic in origin.  So claims Dr. Edward Shorter in an important book, From Paralysis to Fatigue: A History of Psychosomatic Illness in the Modern Era.

Four categories of physical and mental symptoms in which the mind/body fashions diseases states can be identified:
1) Sensory symptoms (e.g., tiredness, burning skin sensations)
2) Motor symptoms (e.g., paralysis)
3) ANS or autonomic nervous system symptoms (e.g., irritable bowel)
4) Psychogenic pain symptoms (e.g., headache caused by psychological factors)

Historically, from 1820 to 1870 the shaping of somatization began with the diagnosis of spinal irritation in which there was cultural shaping of the patient’s symptoms with the physician acting as the agent of culture.  For instance, a condition known as “reflex neurosis” could cause any irritated organ to cause to spread its influence to any other part of the body or brain.  As women were considered the more passive gender at the time, they were considered more susceptible.

Two new models of illness arose after 1870.  One stressed covert but actual disease in the central nervous system (CNS). Neurasthenia or “tired nerves” is a good example:

“…the CNS becomes dephosphorized, or perhaps loses somewhat of its solid constituents; probably also undergoes slight, undetectable, morbid changes in its chemical structure and as a consequence becomes more or less impoverished in the quantity and quality of its nervous force.”

Another stressed the psychological basis of somatization.  In this case, the mind creates actual physical symptoms but the patient accepts them as signs of real physical disease such as the experience of pain:

“From the cultural pool, pain is selected from the symptom pool. But, how people experience pain, how they describe it to others, and how and where they seek help is another thing.”

In the United States, the most common form of somatization is chronic pain syndrome or fibromyalgia.  Its origins have been variously attributed to chronic neurosis, infectious mononucleosis, Epstein-Barr virus, muscle weakness or neuromyasthenia, benign myalgic encephalomyelitis, as well as yeast infections.

The attribution of an illness involves two phases, according to Dr. Shorter: In the first phase, the patient appropriates a real physical disease as a “template” whose actual cause is difficult to substantiate.

In the second phase, the patient broadcasts this “template” to others as an explanation for their set of symptoms.  Broadcasting is abetted by numerous factors including solicitous physicians, pharmaceutical companies that stand to make money from selling prescription drugs and other treatments, patient support groups, and particularly, the mass media, which makes money in increased advertising revenue.

The patient draws upon the pool of symptoms from this culture as models of illness to help them understand their “conversations” with their bodies.  Social isolation and loneliness increase somatization and they are correlated with ill health, actual physical disease, frequent visits to physicians, and somatic complaints.

Some suggested reading:

Sarno, J. E. (1998).  The mindbody prescription: Healing the body, healing the pain. NY: Grand Central Publishing.

Sarno, J. E. (2006).  The divided mind: The epidemic of mindbody disorders. NY: HarperCollins.

Shorter, E. (1992).  From paralysis to fatigue: A history of psychosomatic illness in the modern era. NY: The Free Press.

Tuesday
Apr272010

Obsessive-Compulsive Behavior and Everyday Risk-Taking

The ability to shift from one activity (e.g., doing one’s taxes) to another (e.g., answering the phone) involves inhibiting the first activity to pursue another course of action and results in the production of novel behaviors or novel sequences of behavior. Repetitive stereotyped activities, such as obsessive-compulsive behavior and Gilles de Tourette syndrome, indicate the malfunctioning of this system.  To put it baldly, you’re literally stuck in a cliched set of actions without recourse from your manual tyranny:  Addictive behaviors (gambling, some forms of market investing, drugs, and sex) are one common example, but you also see individuals engaged in stereotypical thinking: About politics, about relationships, about how to carry out a particular procedure at work, about what car to purchase, where to live, what to wear, and so on.  In psychiatry, these are grouped together as OCD (“obsessive-compulsive disorder”) spectrum disorders: Gambling, paraphilia (sexual fetishes), body dysmorphic disorder (e.g., thinking you’re fat when you’re thin), trichotillomania (constant hair-pulling), hypochondriasis, somatization disorder (i.e., frequent psychosomatic complaints), Gilles de Tourette syndrome, autism and Asperger’s syndrome, kleptomania, impulse control disorders, obsessive-compulsive personality disorder, bulimia, and anorexia nervosa.

The cortex (namely, the orbitomedial frontal cortex), the subcortex (particularly a structure known as the “basal ganglia”), and the body, work together in communion with the social and physical environment to accomplish everyday “intellective” tasks.  The orbitomedial frontal cortex is involved in inhibiting (often, socially-inappropriate) behaviors and freeing the mind from distractions to the task at hand.  The basal ganglia, located deep within the subcortex or interior of the brain, dynamically modulates behavior based on feedback from the motor and affective systems and from our various sensory modalities (i.e., touch, vision, audition, and so on).  These two brain structures are intimately connected and cutting their nerve fibers, as it turns out, is sometimes useful with patients with refractory OCD.

Indeed, a malfunctioning basal ganglia leads to stereotyped movement patterns and the absence of novel behaviors.  One patient I treated had obsessive thoughts that he was going to be infected with a sexually transmittable disease from casual sex (OCD) and another I observed in a clinic was a compulsive swearer (Gilles de Tourette syndrome).  The first gentleman was a particularly interesting clinical case because he grew up in a home where his father was a compulsive gambler and continually stole money from his wife and two sons.  As a result, the family was always broke and family finances—like mortgage, electric, and gas payments—were often unmet.  The twin brothers, who were identical twins, both suffered from OCD.  The mother was the only “normal” individual in the family.  It must have been quite difficult for her raising a family of compulsive gamblers and “ideators.”  In both cases, the basal ganglia along with the orbitofrontal cortex, constrained the sons’ ability to switch mental set and each of them was mentally “stuck” in one mode or the other of responding to the world.  The actions of the first sibling led to certain thoughts and the thoughts of the second led to certain actions.

Clomipramine (“Anafranil”), a nonselective serotonin reuptake inhibitor (NSRI), can be an effective treatment for OCD.  However, about 40% of patients with obsessive-compulsive disorder do not respond to either NSRI or selective serotonin reuptake inhibitor (SSRI) treatment.  This may be so because some forms of obsessive-compulsive behavior are a result of excessively high levels of dopamine, not serotonin.  High doses of dopaminergic drugs that increase production of dopamine in the brain (e.g., amphetamine, apomorphine, bromocriptine, and L-DOPA) appear to increase stereotypical movement and compulsive behaviors in humans in one of four dopamine pathways, the nigrostriatal pathway.  On the other hand, another dopamine pathway, the mesocortical pathway, appears to be involved with some of the “cognitive” symptoms of OCD, such as obsessive thoughts.  Genetic studies suggest that these kinds of obsessive-compulsive disorders are highly heritable.  As a result, it is often difficult to treat OCD individuals with supportive psychotherapy alone so treatment is often augmented with the use of psychoactive drugs.

What about everyday risk-taking?  Many forms of gambling and speculation (e.g., investing in stock options and mortgage-backed securities), high-risk activities (such as bungee jumping, rock climbing, cave exploration, and parachuting), and similar pursuits, probably arise from deep roots in human nature that are affected by culture, age (e.g., adolescence), and experience.  They have many positive and some negative benefits and potentially lurk within all of us.  To be sure, a sobering thought.

Some suggesting readings:

American Psychiatric Association.  (2000).  Diagnostic and statistical manual of mental disorders (4th ed., text revision). Washington, D.C.:  American Psychiatric Association.

Horwitz, A. (2002).  Creating mental illness. Chicago: University of Chicago Press.

Stahl, S. M. (2000).  Essential psychopharmacology: Neuroscientific basis and practical applications (2nd. ed.). Cambridge: University of Cambridge Press.